It helps with various diabetes complications and enhances glucose uptake and inhibits glycosylation (the abnormal attachment of sugar to protein).
DIABLINmay not reduce blood sugar levels in healthy, non-diabetic people.
Controls blood sugar levels
Improves the metabolism of glucose and enhances uptake
Reduces the daily insulin requirement of people with Type 1 Diabetes
Protects the membranes of the lens of the eye
Improves diabetic nerve damage
Stimulates insulin production by the pancreas
May also help lower blood triglyceride and cholesterol levels (but does not lower HDL "good" cholesterol levels)
INGREDIENTS:
Fenugreek SeedB.E.E. - contains alkaloids (mainly triganeline), steroidal saponins, mucilaginous fiber and protein high in lysine, L-tryptophan and isoleucine.
It helps stimulate insulin and lower blood sugar levels in people with atherosclerosis and non-insulin-dependent diabetes (Type II) while reducing the daily insulin requirement in people with Type I diabetes.
It also inhibits cholesterol absorption and synthesis.
Siberian Ginseng Root B.E.E. - mainly contains eleutherosidesis and some polysaccharides that help reduce blood sugar levels, increase general resistance to disease and relieve chronic fatigue and stress. It also benefits the immune system.
Onion Bulb (Quercetin)B.E.E. - contains certain sulfur compounds,
such as allyl propyl disulfide (APDS) and flavonoids, including quercetin that helps to block the breakdown of insulin by the liver, and possibly stimulate insulin production by the pancreas, thus increasing the amount of insulin and reducing sugar levels in the blood. It also helps protect the membranes of the lens of the eye from accumulations of polyols as a result of high glucose levels.
Rice Husk
Rice Husk B.E.E.- contains a wide variety of vitamins, minerals, and fibers. It helps reduce the absorption of sugar.
Alpha Lipoic Acid (ALA) - is a vitamin-like antioxidant that is capable of regenerating several other antioxidants back to their active states, including vitamin C, vitamin E, etc. Alpha Lipoic Acid enhances glucose uptake in Type 2 diabetes, inhibits glycosylation and has been used to improve diabetic nerve damage.
SUGGESTED USE:
Start with 1 capsule twice daily, and either increase or decrease the dosage until your blood sugar is controlled. Take DIABLIN with meals.
"Secondary" diabetes refers to elevated blood sugar levels from another medical condition. Secondary diabetes may develop when the pancreatic tissue responsible for the production of insulin is destroyed by disease, such as chronic pancreatitis (inflammation of the pancreas by toxins like excessive alcohol), trauma, or surgical removal of the pancreas.
Diabetes can also result from other hormonal disturbances, such as excessive growth hormone production (acromegaly) and Cushing's syndrome. In acromegaly, a pituitary gland tumor at the base of the brain causes excessive production of growth hormone, leading to hyperglycemia. In Cushing's syndrome, the adrenal glands produce an excess of cortisol, which promotes blood sugar elevation.
In addition, certain medications may worsen diabetes control, or "unmask" latent diabetes. This is seen most commonly when steroid medications (such as prednisone) are taken and also with medications used in the treatment of HIV infection (AIDS).
What are the different types of diabetes? Type 1 Diabetes | Type 2 Diabetes |Gestational Diabetes | Secondary Diabetes Finally, diabetes occurs much more frequently in women with a prior history of diabetes that develops during pregnancy called gestational diabetes.
Gestational Diabetes - can occur temporarily during pregnancy. Significant hormonal changes during pregnancy can lead to blood sugar elevation in genetically predisposed individuals. Blood sugar elevation during pregnancy is called gestational diabetes. Gestational diabetes usually resolves once the baby is born. However, 25%-50% of women with gestational diabetes will eventually develop type 2 diabetes later in life, especially in those who require insulin during pregnancy and those who remain overweight after their delivery. Patients with gestational diabetes are usually asked to undergo an oral glucose tolerance test about six weeks after giving birth to determine if their diabetes has persisted beyond the pregnancy, or if any evidence (such as impaired glucose tolerance) is present that may be a clue to the patient's future risk for developing diabetes.
Type 2 diabetes was also referred to as non-insulin dependent diabetes mellitus (NIDDM), or adult onset diabetes mellitus (AODM). In type 2 diabetes, patients can still produce insulin, but do so relatively inadequately for their body's needs, particularly in the face of insulin resistance as discussed above.
In many cases this actually means the pancreas produces larger than normal quantities of insulin. A major feature of type 2 diabetes is a lack of sensitivity to insulin by the cells of the body (particularly fat and muscle cells).
In addition to the problems with an increase in insulin resistance, the release of insulin by the pancreas may also be defective and suboptimal. In fact, there is a known steady decline in beta cell production of insulin in type 2 diabetes that contributes to worsening glucose control. (This is a major factor for many patients with type 2 diabetes who ultimately require insulin therapy.)
Finally, the liver in these patients continues to produce glucose through a process called gluconeogenesis despite elevated glucose levels. The control of gluconeogenesis becomes compromised.
While it is said that type 2 diabetes occurs mostly in individuals over 30 years old and the incidence increases with age, we are seeing an alarming number patients with type 2 diabetes who are barely in their teen years. In fact, for the first time in the history of humans, type 2 diabetes is now more common than type 1 diabetes in childhood. Most of these cases are a direct result of poor eating habits, higher body weight, and lack of exercise.
While there is a strong genetic component to developing this form of diabetes, there are other risk factors - the most significant of which is obesity. There is a direct relationship between the degree of obesity and the risk of developing type 2 diabetes, and this holds true in children as well as adults. It is estimated that the chance to develop diabetes doubles for every 20% increase over desirable body weight.
Regarding age, data shows that for each decade after 40 years of age regardless of weight there is an increase in incidence of diabetes. The prevalence of diabetes in persons 65 to 74 years of age is nearly 20%.
Type 2 diabetes is also more common in certain ethnic groups. Compared with a 6% prevalence in Caucasians, the prevalence in African Americans and Asian Americans is estimated to be 10%, in Hispanics 15%, and in certain Native American communities 20% to 50%.
There are two major types of diabetes, called Type 1 and Type 2.
Type 1 diabetes was also called insulin dependent diabetes mellitus (IDDM), or juvenile onset diabetes mellitus.
In Type 1 diabetes, the pancreas undergoes an auto-immune attack by the body itself, and is rendered incapable of making insulin. Abnormal antibodies have been found in the majority of patients with type 1 diabetes.
Antibodies are proteins in the blood that are part of the body's immune system.
The patient with Type 1 diabetes must rely on insulin medication for survival. In auto-immune diseases, such as type 1 diabetes, the immune system mistakenly manufactures antibodies and inflammatory cells that are directed against and cause damage to patients' own body tissues.
In persons with type 1 diabetes, the beta cells of the pancreas, which are responsible for insulin production, are attacked by the misdirected immune system. It is believed that the tendency to develop abnormal antibodies in type 1 diabetes is, in part, genetically inherited, though the details are not fully understood.
Exposure to certain viral infections (mumps and Coxsackie viruses) or other environmental toxins may serve to trigger abnormal antibody responses that cause damage to the pancreas cells where insulin is made. Some of the antibodies seen in type 1 diabetes include anti-islet cell antibodies, anti-insulin antibodies and anti-glutamic decarboxylase antibodies. These antibodies can be measured in the majority of patients, and may help determine which individuals are at risk for developing type 1 diabetes.
At present, the American Diabetes Association does not recommend general screening of the population for Type 1 diabetes, though screening of high risk individuals, such as those with a first degree relative (sibling or parent) with Type 1 diabetes should be encouraged.
Type 1 diabetes tends to occur in young, lean individuals, usually before 30 years of age, however, older patients do present with this form of diabetes on occasion. This subgroup is referred to as latent auto-immune diabetes in adults (LADA).
LADA is a slow, progressive form of Type 1 diabetes. Of all the patients with diabetes, only approximately 10% of the patients have Type 1 diabetes and the remaining 90% have Type 2 diabetes.
What are the symptoms of diabetes?
The early symptoms of untreated diabetes are related to elevated blood sugar levels, and loss of glucose in the urine. High amounts of glucose in the urine can cause increased urine output and lead to dehydration. Dehydration causes increased thirst and water consumption.
The inability of insulin to perform normally has effects on protein, fat and carbohydrate metabolism. Insulin is an anabolic hormone, that is, one that encourages storage of fat and protein. A relative or absolute insulin deficiency eventually leads to weight loss despite an increase in appetite. Some untreated diabetes patients also complain of fatigue, nausea and vomiting. Patients with diabetes are prone to developing infections of the bladder, skin, and vaginal areas. Fluctuations in blood glucose levels can lead to blurred vision. Extremely elevated glucose levels can lead to lethargy and coma.
How is diabetes diagnosed? The fasting blood glucose (sugar) test is the preferred way to diagnose diabetes. It is easy to perform and convenient. After the person has fasted overnight (at least 8 hours), a single sample of blood is drawn and sent to the laboratory for analysis. This can also be done accurately in a doctor's office using a glucose meter. Normal fasting plasma glucose levels are less than 100 milligrams per deciliter (mg/dl). Fasting plasma glucose levels of more than 126 mg/dl on two or more tests on different days indicate diabetes.
A random blood glucose test can also be used to diagnose diabetes. A blood glucose level of 200 mg/dl or higher indicates diabetes. When fasting blood glucose stays above 100mg/dl, but in the range of 100-126mg/dl, this is known as impaired fasting glucose (IFG). While patients with IFG do not have the diagnosis of diabetes, this condition carries with it its own risks and concerns, and is addressed elsewhere.